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. 2023 Jun 13;9(6):e16865. doi: 10.1016/j.heliyon.2023.e16865

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© 2023 The Authors

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 5 — Effects of regulating DSCAS on the sensitivity of cisplatin to lung cancer cells. (A). Cell cycle assay displayed that the proportion of G0/G1 phase cells in sh-DSCAS group decreased more than that in sh-NC group, and the proportion of G0/G1 phase cells in oe-DSCAS group decreased less than that in oe-NC group. (n = 3, P < .05). (B). CCK-8 for cell proliferation-toxicity assay indicated cisplatin IC50 of SK-MES-1 harboring oe-DSCAS was highest (37.20 μM), while cisplatin IC50 of SK-MES-1 harboring sh-DSCAS was lowest (15.30 μM). (n = 3, P < .05). *P < .05,**P < .01,***P < .001，****P < .0001 compared with control.