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. 2023 Jun 12;13:1183318. doi: 10.3389/fonc.2023.1183318

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Copyright © 2023 Zerella, Homan, Arts, Brown, Scott and Hahn

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Gene Ontology of TFs implicated in hematological development and disease. Using ShinyGO0.77 analysis software (31), the known BMF/HM predisposition list in Table 1 and the potential list of BMF/HM predisposition genes in Table 4 (including ERG, GATA3 and SPI1) were analyzed for enrichment of GO terms in biological process and Molecular functions using all known human TF genes as background (i.e., normalization). All protein coding genes were used for background in GO cellular component analysis as there was no enrichment when using TFs as background.