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. 2023 Jun 20;120(26):e2301360120. doi: 10.1073/pnas.2301360120

Fig. 1.

Fig. 1.

Enzymatic activity of sEH was specifically altered in the liver following TBI. (A) Relative sEH activity in the liver, kidney, heart, spleen, lung, and injured cortex was examined at different time points following CCI in adult male C57BL/6J mice (n = 6). (B) The relative mRNA levels of Ephx2 in the liver at different time points after CCI. (C and D) Western blotting (Left) and quantification (Right) of sEH in the liver (C), and injured cortex (D), at different time points after CCI (n = 6). (E) Relative sEH activity of the liver, kidney, heart, spleen, lung, and injured cortex at different time points following CHI in adult male C57BL/6J mice (n = 6). (F) Western blotting (Left) and quantification (Right) of sEH in the liver and injured cortex at different time points after CHI (n = 6). Statistical significance was determined using one-way ANOVA with the Dunnett's post-hoc test or Brown–Forsythe and Welch ANOVA followed by the Dunnett's T3 post-hoc test. Data are presented as the means ± SEM. Asterisks indicate significant differences (*P < 0.05, **P < 0.01, ***P < 0.001).