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. 2023 Jun 26;21:146. doi: 10.1186/s12915-023-01647-6

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — The effect of dopamine on the retinal clock involves both D1- and D2-like receptors. Means of the endogenous period, the phase and the amplitude of P8 retinal explants, supplemented with DA (n = 7, 50 µM), a D1R DA antagonist (SCH39166, n = 6), a D2R DA antagonist (L741626, n = 6), DA combined to SCH39166 (n = 6) or L741626 (n = 6), or a general gap junction blocker (CBX, n = 4) and compared to non-supplemented control retinas (C, n = 10). Bars represent mean ± SEM (* = p ≤ 0.05; ** = p ≤ 0.01; *** = p ≤ 0.001, comparison with C group; ^## = p ≤ 0,01; ^### = p ≤ 0,001, comparison with DA group)