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. 2023 Jun 16;15(6):1752. doi: 10.3390/pharmaceutics15061752

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© 2023 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Anti-septic effects of T. ni cecropin in an LPS-induced endotoxemia septic shock mouse model. Suppressive effects on circulating serum (a) endotoxin levels and (b) IL-6 in the endotoxemia mouse model. (c–e) Recovery of aspartate aminotransferase (AST), alanine aminotransferase (ALT), and blood urea nitrogen (BUN) levels in the serum of the LPS-stimulated endotoxemia mouse model following treatment with T. ni cecropin. Data are presented as the mean ± SEM (n = 3 per group). ** p < 0.01 and *** p < 0.001 compared to that in the control group (one-way ANOVA). (f) Micrograph of lung histology for control, T. ni cecropin, LPS, and pre-treatment T. ni cecropin groups (clockwise) as indicated at the bottom of each photograph. Bars: 100 μm; 20× magnification.

Anti-septic effects of T. ni cecropin in an LPS-induced endotoxemia septic shock mouse model. Suppressive effects on circulating serum (a) endotoxin levels and (b) IL-6 in the endotoxemia mouse model. (c–e) Recovery of aspartate aminotransferase (AST), alanine aminotransferase (ALT), and blood urea nitrogen (BUN) levels in the serum of the LPS-stimulated endotoxemia mouse model following treatment with T. ni cecropin. Data are presented as the mean ± SEM (n = 3 per group). ** p < 0.01 and *** p < 0.001 compared to that in the control group (one-way ANOVA). (f) Micrograph of lung histology for control, T. ni cecropin, LPS, and pre-treatment T. ni cecropin groups (clockwise) as indicated at the bottom of each photograph. Bars: 100 μm; 20× magnification.