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. 2023 May 27;5(3):fcad169. doi: 10.1093/braincomms/fcad169

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© The Author(s) 2023. Published by Oxford University Press on behalf of the Guarantors of Brain.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (https://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Flow chart of patients indicating the observed clinical phenotypes. ^aγ-aminobutyric acid type B receptor- and Sox1-associated limbic encephalitis (n = 1), multifocal motor neuropathy (n = 1), critical illness neuropathy (n = 1), and seronegative rapidly progressive cerebellar ataxia (n = 1); ^bDiplopia (n = 1), muscle weakness (n = 1), myalgias (n = 1) and paresthaesia (n = 1); ^cAlcohol-related dementia (n = 2), Alzheimer disease (n = 2), arthralgias (n = 1), diabetes-related carpal tunnel syndrome (n = 1), clivus chondroma (n = 1), neurological functional disorder (n = 3), multifactorial gait disorder (n = 1), Parkinson disease (n = 1), primary headache (n = 1), sepsis-associated encephalopathy (n = 3) and steroid myopathy (n = 2). Abbreviations: CIP, chronic intestinal pseudo-obstruction; LEMS, Lambert–Eaton myasthenic syndrome; MG, myasthenia gravis; NMJ, neuromuscular junction; NMT, neuromyotonia.