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Model of acne pathologic processes induced by C. acnes interactions with sebocytes, keratinocytes, and monocytes. Reprinted with permission from Lee et al. [24]. EPS extracellular polymeric substances, CAMP cyclic adenosine monophosphate, TLR toll-like receptor, IL interleukin, TNF tumor necrosis factor, LTB leukotriene B, CD36 cluster of differentiation 36, GM-CSF granulocyte-macrophage colony-stimulating factor, hBD human β-defensin, MMPs matrix metalloproteinases
