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Response to statin, ezetimibe, and proprotein convertase subtilisin/kexin type 9 (PCSK9) inhibitors is dependent on the degree of residual low-density lipoprotein receptor activity; however, for lomitapide, angiopoietin-like protein 3 (ANGPLT3)-directed therapy and lipoprotein apheresis, response is independent of low-density lipoprotein receptor function. Reproduced with permission from Mohamed et al.44.
