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. 2023 Jun 26;26(7):1208–1217. doi: 10.1038/s41593-023-01361-0

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Extended Data Fig. 9 — (a-b) Breakdown of pathways in SMS data that are associated with ASD and neurotypical controls. (c-d) Breakdown of pathways in RNAseq data that are associated with ASD and neurotypical controls. e) Overlap of ASD associated KEGG enzymes derived from the multi-cohort cross-sectional analysis and KEGG enzymes that are found to be present in the microbes that decreased in the Kang et al FMT study. f) Pathway break down of KEGG enyzmes found in both the Kang et al FMT study and ASD children in the multi-cohort cross-sectional analysis. Only microbes that were also found in the SMS data were considered in the Kang et al study.