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. 2023 Jan 21;17(7):1356–1378. doi: 10.1002/1878-0261.13370

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© 2023 The Authors. Molecular Oncology published by John Wiley & Sons Ltd on behalf of Federation of European Biochemical Societies.

This is an open access article under the terms of the http://creativecommons.org/licenses/by/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Fig. 2 — SALL4 expression and its transcriptional signature are characterized by stromal pathways in pancreatic ductal adenocarcinoma. (A) Venn diagram displaying the number of genes correlated with SALL4 expression that was intersected between the ICGC, TCGA, and GSE85916 cohorts. Heatmap visualizing the relative average expression of indicated genes (rows) according to SALL4 expression for 259 patients with localized pancreatic carcinoma in the ICGC cohort. (B) Kaplan–Meier curves of overall survival according to 24‐genes signature expression (best cutoff), compared using the log‐rank test, in ICGC and GSE85916 cohorts. (C) Over‐representation analysis (ORA) of genes correlated with SALL4 expression from KEGG pathways and GeneOntology (biological process; FDR < 0.01).