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. 2023 Jul 8;14(7):408. doi: 10.1038/s41419-023-05938-2

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — A Representative images of UBE2S and LPP immunohistochemistry staining in BCa tissues. Scale bars: black, 200 μm; red, 50 μm. B Pearson’s correlation analysis of UBE2S and LPP protein expression in Cohort 2, which consisted of 59 paired BCa tissues. C LPP protein levels in Cohort 2. NAT normal adjacent tissues; LN− CA, BCa tissues without lymphatic metastasis; LN+ CA, BCa tissues with lymphatic metastasis. D Relative LPP mRNA expression in NAT and BCa tissues from the TCGA database. Kaplan‒Meier curves showing the overall survival (E) and disease-free survival (F) of BCa patients with high LPP expression and low LPP expression in cohort 2. G LPP protein levels in LPP-silenced BCa cells, as detected by western blot assays. Representative images and quantitative analysis of wound healing (H), transwell migration (I) and invasion (J) assays using LPP-silenced BCa cells. Representative images and quantitative analyses of wound healing (K), transwell migration (L) and invasion (M) assays of UBE2S-silenced BCa cells combined with LPP knockdown. *P < 0.05 and **P < 0.01.