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. Author manuscript; available in PMC: 2024 Aug 15.
Published in final edited form as: Environ Res. 2023 May 22;231(Pt 2):116213. doi: 10.1016/j.envres.2023.116213

Figure 1.

Figure 1.

Prenatal PAH exposure lowered Pparγ methylation among grandoffspring (F2) mice at PND28, but not among offspring (F1) mice at PND28. Percent CpG methylation in mammary tissue of three sites (CpG-189, CpG-205, and CpG-241) in the Pparγ promoter region are shown following control (open circles) and PAH exposure (open squares) among F1 PND28 (control n = 7; PAH n = 10) and F2 mice at PND28 (control n = 10; PAH n = 10). Mean and 95% confidence intervals are marked. The p values for the association between prenatal PAH exposure and Pparγ methylation are shown for significant and borderline significant associations. Analyses were performed by t test. Abbreviations: PND: postnatal day, F1: first generation offspring, F2: second generation offspring.