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. 2023 Jun 30;14(3):1412–1433. doi: 10.21037/jgo-23-277

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2023 Journal of Gastrointestinal Oncology. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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Erastin or sorafenib suppressed AKR1C1 expression in HCC cells. (A) GSE104462 shows that erastin suppressed AKR1C1 expression in HepG2 cells. (B) Different concentrations of erastin were co-cultured with LEN-HepG2 cells for 24 h. According to the results, erastin suppressed AKR1C1 expression in LEN-HepG2 cells. (C) Sorafenib suppressed AKR1C1 expression in LEN-HepG2 cells after being co-cultured for 24 h. *P<0.05, **P<0.01, ***P<0.001, ****P<0.0001. NC, negative control.