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. 2023 Apr 7;43(6):2643–2673. doi: 10.1007/s10571-023-01344-6

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — Pathways for neural stem cell transplantation and mechanisms for restoring cognitive dysfunction. Transplantation of neural stem cells triggers endogenous synaptogenesis and endogenous neurogenesis, affecting recovery of cognitive impairment. The limited causal relationship between amyloid-beta (dashed arrows) and neural stem cells rules out any link between behavior and amyloid-beta aggregation