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The mechanism of development of NSAID-associated gastropathy. 1—disrupted phospholipid monolayer, 2—damage to tight junction proteins, 3—uncoupled mitochondria, 4—pronounced regenerative changes in the epithelium (foveolar hyperplasia with hyperchromic cell nuclei, decreased mucus formation), 5—subnuclear vacuolated mucous cells, 6—mild diffuse mononuclear infiltration, 7—bundles of smooth muscle cells in the lamina propria, 8—edema with ectatic blood vessels.
