Table 2.

Variation, loss, and gain of body weight (g) of Wistar rats with inflammation induced by the HGLI diet; adults (31 weeks) on the first and last day of the experiment, submitted to different treatments after ten days of investigation.

Groups	∆Weight (g) Mean (SD)	Average Weight Loss (g)	Average Weight Gain (g)
No treatment	1.40 (9.61) a	8.00 (1.41)	7.70 (6.03)
Conventional treatment	−3.00 (10.46) a	11.00 (8.49)	5.00 (0.00)
Test treatment 1 (CE)	−5.20 (11.82) a	12.30 (9.29)	5.50 (2.12)
Test treatment 2 (EPG)	−11.00 (8.40) a	11.00 (8.40)	-

No treatment: HGLI diet + 1 mL of water by gavage; conventional treatment: nutritionally adequate diet (Labina^® feed) + 1 mL of water per gavage; test treatment 1: HGLI diet + 1 mL of CE at a concentration of 12.5 mg/kg by gavage; test treatment 2: HGLI diet + 1 mL of EPG at a concentration of 50 mg/kg by gavage; HGLI diet: mixture composed of Labina^®, condensed milk and sugar (1:1:0.21 w/w/w); HGLI: high glycemic index and high glycemic load diet; CE: crude extract rich in carotenoids from Cantaloupe melons; EPG: crude extract rich in carotenoids from Cantaloupe melons nanoencapsulated in porcine gelatin. The values obtained for ∆ body weight (final body weight − initial body weight) were expressed as mean (standard deviation). The average weight loss and gain (g) results were expressed as mean (standard deviation) based on the number of animals from the same group that lost or gained weight. Equal letters in the same column indicate no significant difference between the groups evaluated for each parameter (p > 0.05). For the evaluation of data normality, the Kolmogorov–Smirnov test was used. Variations in body weight (∆) had a non-parametric distribution, so the Kruskal–Wallis test and Dunn’s post-test were used to verify differences between the evaluated groups. Values of p ≤ 0.05 were considered statistically significant.