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. 2023 Jul 12;9(8):e1500. doi: 10.1097/TXD.0000000000001500

TABLE 1.

Correlation of serum DSA with histology (gray shows de novo DSA)

No. Preformed DSA DSA test days after SLKT Post-DSA Bx-days after SLKT C4d kidney Kidney path summary C4d liver Liver path summarya Outcome
Class I Class II Class I Class II
1 0 0 65 0 9000 65, 66 Focal ptc positivity Mild acute AMR (g1-2, i1, t0, v0, ptc1); mild to moderate chronic changes (cg0, ci1, ct1, cv2) likely related to donor disease Diffuse portal microvascular endothelial cell positivity; negative LSEC; focal KC cytoplasmic positivity Low-grade acute AMR (i3, h2) with prominent KC hypertrophy with KC C4d staining Renal allograft failure 3. 5 mo; biliary strictures developed; death 9 y post-SLKT
2 6700 0 13 0 0 15, 13 Diffuse ptc positivity Mild acute AMR (g1-2, i1, t0, v0, ptc2); no chronic changes (cg0, ci0, ct0, cv0) likely related to donor disease Diffuse portal microvascular endothelial cell positivity; negative LSEC Low-grade AMR (i3, h2); KC less prominent than case no. 1 Death 8 y post-SLKT from liver allograft failure
3a 23 000b 25 000 54 10, 54 Diffuse ptc positivity Mild AMR (g1, i1, t0, ptc1); no chronic changes (cg0, ci0, ct0, cv0, ah0) Diffuse portal microvascular and central vein endothelial cell positivity; negative LSEC Mixed TCMR and AMR (i3, h2) with central perivenular and sclerosing central vein lesions in regions of prominent C4d deposition Renal allograft failure 2. 5 y posttransplant: HAT and death 11 y post-SLKT
3b 301 15 000 18 000 301 346 Minimal ptc positivity Suspicious for AMR (g0, I1, t0, v0, ptc1); mild chronic changes (cg0, ci1, ct1, cvo, ah0) Negative Negative for TCMR and AMR with intact architecture with periportal-shunt-type vessel
4a 13 000 0 5 NA NA 8, 5 Negative No specific histopathologic changes (go, i0, t0, v0, ptc0); no chronic changes (cg0, ci0, ct0, cv0, ah0) Minimal microvascular endothelial cell positivity Minimal changes (i1, h0) with prominent KC hypertrophy GFR at 3 y 36 mL/min; follow-up liver biopsy showed plasma cell rich TCMR and chronic AMR; death from a GI bleed 4. 5 y post-SLKT
4b 74, 134 Minimal ptc positivity Minimal histopathologic changes/suspicious for minimal AMR (g0, i1, t0, vo, ptc1); mild chronic changes (cg0, ci1, ct1, cv0, ah1) Minimal portal capillary and LSEC positivity Plasma cell-rich TCMR with interface activity and central perivenulitis/probably chronic AMR (i2, h2)

aScoring per Banff 2016 criteria.

bC1q positive DSA.

AMR, antibody-mediated rejection; Bx, biopsy; DSA, donor specific alloantibodies; GFR, glomerular filtration rate; GI, gastrointestinal; HAT, hepatic artery thrombosis; KC, Kupffer cell; LSEC, liver sinusoidal endothelial cell; NA, not available; ptc, peritubular capillary; SLKT, simultaneous liver-kidney transplant; TCMR, T-cell mediated rejection.