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. 2023 Jul 19;9(29):eadg5358. doi: 10.1126/sciadv.adg5358

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Fig. 1. — (A) Preparation of MSCs-Lip@NCAF. PC, phosphatidylcholine. (B) MSCs-Lip@NCAF migrated to injured lungs because of their homing ability, and Lip@NCAF was released in response to MMP-2. Then, Lip@NCAF degraded collagen fibers and inhibited fibroblast overactivation. Moreover, MSCs repaired injured AEC IIs in young mice and differentiated into AEC IIs to participate in alveolar reestablishment in aged mice.