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. 2023 Jul 20;14:4383. doi: 10.1038/s41467-023-40093-4

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a Arabidopsis chc2-1 and ap2σ mutants exhibited enhanced disease resistance to B. cinerea infection. b, c INTAM≫RFP-HUB1(b) and XVE≫AX2 (c) transgenic plants showed enhanced resistance phenotype to B. cinerea infection after induction by 2 μM 4-Hydroxytamoxifen or 5 μM estradiol, respectively. d Arabidopsis flot1/2 double mutants showed increased susceptibility to B. cinerea infection. e Arabidopsis rem1.2 and rem1.3 mutants exhibited similar susceptibility to B. cinerea compared with the wild-type. Relative lesion size was determined by comparing lesion size to the size of the entire leaf 2 days after infection. The data are presented as mean ± s.d., n = 10 biological replicates. The statistical analysis in b and c was performed using unpaired two-tailed Student’s t-test. The statistical analysis in a, d and e were performed using ANOVA Dunnett’s multiple comparisons test. The small open circles represent individual values. The error bars indicate s.d. *P < 0.05, **P < 0.01, ***P < 0.001. Source data are provided as a Source Data file.