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. 2023 Mar 15;18(11):2497–2503. doi: 10.4103/1673-5374.371379

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Copyright: © Neural Regeneration Research

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Schematic illustration of therapeutic effects of RF-EMFs exposure on the brain of AD mice.

(A) AD-associated morphological changes of microglia and microglial-related genes. (B) Proposed mechanism of RF-EMFs action on AD. In the brain of AD mice, microglial dysfunction and neuroinflammation are induced, which result in disturbances of memory processing. In activated microglia of AD mice, the expression levels of microglial-related genes (Trem2, Fcgr1, Spi1, and Ctss) and pro-inflammatory genes are up-regulated. RF-EMFs exposure has the potential to positively restore cognitive impairments in AD mice via the modulation of microglial-related and neuroinflammation-related genes. AD: Alzheimer’s disease; IL: interleukin; RF-EMFs: radiofrequency electromagnetic fields.