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. 2023 Jun 7;9(6):e16963. doi: 10.1016/j.heliyon.2023.e16963

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© 2023 The Authors

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 6 — Histopathology of liver and kidney sections of the animals treated with both PG at different concentrations A1 (10 mg/kg), A2 (100 mg/kg), A3 (1000 mg/kg), A4 (2900 mg/kg), A5 (5000 mg/kg) and A6 (Normal saline). Liver sections of groups treated with PG-PNPs; B1 (10 mg/kg), B2 (100 mg/kg), B3 (1000 mg/kg), B4 (2900 mg/kg), B5 (5000 mg/kg) and B6 (control). Kidney sections of groups treated with PG; A1 (10 mg/kg), A2 (100 mg/kg), A3 (1000 mg/kg), A4 (2900 mg/kg), A5 (5000 mg/kg) and A6 (control). Kidney sections of groups treated with PG-PNPs; B1 (10 mg/kg), A2 (100 mg/kg), B3 (1000 mg/kg), B4 (2900 mg/kg), B5 (5000 mg/kg) and B6 (control). [H and E x 40].