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. 2023 Jul 21;18:204. doi: 10.1186/s13023-023-02804-4

Table 2.

Clinical characteristics of the untreated versus treated groups

Non-miglustat group n = 16 Miglustat-treated group n = 10 p value
Year of birth
 1987–2005 15 0
 2006–2012 1 10
Sex ratio ( boys/girls) 11/5 5/5 0.42
NP-C1 (NPC1 gene) 14 10 0.51
NP-C2 (NPC2 gene) 2 0
Consanguinity 44% (7/16) 50% (5/10)
Age at diagnosis (years) 1.88 y (antenatal-4.9) 0.3 y (0.04–1.4) 0.02
Age at neurological onset (months) 12 m (3–18) 9 m (5–18) 0.22
Diagnosis before neurological onset 37.5% 100% 0.003
Age at death (years) 11/16 4.42 (2.58–6.52) 10/10 5.56 (2.79–7.43) 0.16
Visceral signs
 Neonatal cholestasis 5/16 (31%) 9/10 (90%) 0.005
 Duration (months) 3 m (3–6); uk n = 2 8 m (3–9); uk n = 3 Nd
 Specific pulmonary dysfunction 5/12 (42%) 6/10 (60%) 0.70
 Splenomegaly 13/14 (93%) 8/8 (100%)  > 0.99
 Age at discovery (months) 1.4 m (0.01–28) 1 m (0.1–4) 0.20
 Hepatomegaly 12/13 (92%) 9/9 (100%)  > 0.99
 Age at discovery (months) 3.2 m (0.4–33) 1 m (0.1–4) 0.07
Nutrition
 Malnutrition at last follow-up 4/13 (31%) 2/8 (25%) 1
 Nutritional support 8/9 (88%) 8/8 (100%)  > 0.99
 Age at start (years) 2.75y (1.5–6) 2.92y (0.5–6.7) 0.92
Medications
 Oxygen or non-invasive ventilation 2/7 (29%) 7/9 (78%) 0.13
 Antiepileptics 5/7 (71%) 4/8 (50%) 0.61
 Antispastics or antidystonics 6/7 (86%) 7/10 (70%)  > 0.99
 Antalgics 5/7 (71%) 6/7 (86%)  > 0.99

Percentages were calculated taking into account the number of patients for whom information was available. Results are reported as median (range). Abbreviations: y = years; m = months; uk = data unknown; nd = no statistical test due to inadequate sample size