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Model of post-translational regulation of GI abundance by HOS15 and COP1.
HOS15 represses flowering by promoting low ambient temperature–mediated degradation of GI protein, a floral activator. Low ambient temperature (16°C) promotes the dissociation of HOS15 and COP1, allowing HOS15 and COP1 to play independent roles in low ambient temperature-induced GI turnover. Therefore, FT expression is reduced and flowering is delayed under low ambient temperature.
