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. 2023 Jul 23;13:11890. doi: 10.1038/s41598-023-38976-z

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Results of a histological examination of the experimental animals: (A) liver, control group: normal structure of liver, 100 × magnification; (B) liver, exposure group 2: pronounced dystrophic and necrobiotic changes in hepatocytes, 100 × magnification; (C) the increased number of prokaryotic hepatocytes, %, * p < 0.05 compared with controls; (D) kidneys, control group: the epithelium of convoluted tubules of kidneys with a clear PAS-positive brush border on the apical edge, the cytoplasm is homogeneous, the nuclei are well visualized, the lumens of the tubules are not dilated, 100 × magnification; (E): kidneys, exposure group 2: foci of destruction of the PAS-positive brush border of the tubular epithelium, dystrophic changes in the cytoplasm, tubular luminal dilation, 100 × magnification; (F) loss of the brush border of the renal tubular epithelium, %, * p < 0.05 compared with controls.