Fig. 3.

The illustration shows the effects of antioxidants on miRNA expression during exposure to hazardous compounds. (A) CAPE could downregulate hepatotoxicity induced by CdCl2 through upregulation of miR-182-5p. (B) Selenium in common carp lymphocytes may inhibit the promotion of miR-216a, necrosis, and apoptosis caused by CdCl2. (C) Quercetin, via inhibiting miR-21, could attenuate liver fibrosis and steatosis induced by cadmium. (D) Selenium yeast in the kidney of the chicken could protect against necroptosis caused by CdCl2 by increasing the expression of miR-26a-5p. (E) N-acetylcysteine in male albino rats could attenuate Cd-induced hepatotoxicity by decreasing the expression of miR-146a and inflammation.