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. 2023 Jun 26;4(7):984–1000. doi: 10.1038/s43018-023-00584-1

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Extended Data Fig. 5 — a) Phylogenetic sample trees of Cluster#1 (top left) and Cluster#2 (bottom left) RCC tumor-bearing mice; different pattern of evolution can be clearly appreciated. Oncoprints of Cluster#1 (top right) and Cluster#2 (bottom right) RCC tumor-bearing mice displaying time of driver engineered and spontaneous somatic events in primary and secondary lesions.

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