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. 2023 Jun 26;4(7):984–1000. doi: 10.1038/s43018-023-00584-1

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Extended Data Fig. 3 — a) Trinucleotide context-specific somatic SNV frequencies as detected by WGS in 2 metastatic samples (upper panel), 6 primary tumors and 2 cell line samples (lower panel) as compared to corresponding trinucleotide context-specific somatic SNV frequencies in pan-kidney tumor cohort (n = 148 tumors) analyzed by WGS. b-c) Comprehensive SM-GEMM cohort frequency plot showing percentage of double base substitutions (b) and indels (c) as calculated from WES data analysis.