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. 2023 Jul 25;6:775. doi: 10.1038/s42003-023-05143-z

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a CCK-8 assay showed HT29 cell proliferation after transfected with miR-182-5p mimic, mimic control, inhibitor, or inhibitor control. b Colony formation assay of HT29 cells after transfected with miR-182-5p mimic, mimic control, inhibitor, and inhibitor control. c CCK-8 assay showed SW480 cells proliferation after transfection. d Colony formation assay of SW480 cells after transfection. e MiR-182-5p mimic could significantly reduce NAD+ level and inhibitor of miR-182-5p led to higher NAD+ level in HT29 cells. f MiR-182-5p inhibitor could significantly increase NAD+ level in HT29 NFIB knockdown cells. Bar graph data are presented as the mean ± SD; *P < 0.05; **P < 0.01; ***P < 0.001.