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. 2023 Jul 26;23:699. doi: 10.1186/s12885-023-11045-4

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — Dose–response of SNU449, 1BR-hTERT, HepG2 and SKOV3, and upon 37-mer peptide treatment. Cell response of each specific cell type was normalized to a scale running from 0 to 100. A Peptide treatment response of SNU449. IC50 value was calculated as 76.4 ± 0.6015 μM (R2 = 0.8410) from 24 h treatment, and 88.4 ± 0.9148 μM (R2 = 0.8969), from 48 h exposure, respectively. B Cisplatin exposure of SNU449 at 48 h produced a response curve from which the IC50 value was calculated as 12.79 ± 2.055 μM (R2 = 0.7716). C 1BR-hTERT 24 h dose–response curve generated an IC50 of 90.5 ± 0.521 μM (R2 = 0.9481). D HepG2 peptide 24 h treatment response curve produced an IC50 of 33.65 ± 1.09 μM (R2 = 0.8528). E SKOV3 response from 24 h peptide treatment produced an IC50 value of 27.45 ± 1.5085 μM (R2 = 0.8404)