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. 2023 Jul 14;17:1225191. doi: 10.3389/fnins.2023.1225191

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Copyright © 2023 McGuigan, Marie, O'Bryan, Flores, Evered, Silbert and Scott.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Potential mechanisms of xenon induced neuroprotection. Xenon influences multiple overlapping cellular processes that promote cell survival. Green Xe = xenon increases protein expression/ionic current, Red Xe = xenon reduces protein expression/ionic current, Green protein = pro-survival protein, Red protein = pro-apoptosis protein. Akt, Ak strain transforming protein; Bcl-2, B-cell lymphoma protein; Bax, Bcl-2-associated X protein; BDNF, Brain derived neurotrophic factor; CREB, cAMP response element binding protein; Cyt-c, Cytochrome c; GSK3β, Glycogen synthase kinase 3 beta; Hmox1, Heme oxygenase 1; MAPK, Mitogen activated protein kinase; mTOR, Mammalian target of rapamycin; NMDA, N-methyl-D-aspartatel; PKC, Protein Kinase C. Created with BioRender.com.