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Consequences and mechanisms of direct and indirect tyrosine kinase inhibitor (TKI)-induced cardiotoxicity. ER, endoplasmic reticulum; IHD, ischemic heart disease; LV/RV, left ventricle/right ventricle; LVSD, left ventricular systolic dysfunction; ROS, reactive oxygen species; SVT, supraventricular tachycardia. (Created with BioRender.com.)
