(A) A schematic diagram illustrating the knock-in strategy for generating Slc6a17-HA-2A-iCre mice. More details in Figure 1—figure supplement 1D. (B) Higher magnification views of co-immunostaining by anti-Syp and anti-HA antibodies in the hippocampus. Scale bar = 10 μm. (C) Immunoisolation of SLC6A17-HA containing vesicles by the anti-HA antibody coated on magnetic beads. SLC6A17-HA fraction was positive for Syp, Syt1, Syb2, v-ATPase, VGluT1, VGluT2, and vGAT, but negative for GluT4 and transferrin receptor. (D) Further purification of the LP2 fraction by sucrose gradient showed that SLC6A17-HA was co-immunoisolated with Syp, Syt1, and VGluT1, but not PSD95, ERp72, EEA1, SNAP23, PSMC6, or STX6. (E) A schematic diagram illustrating the APEX2-based labeling strategy with AAV-PHP.eb virus mediated SLC6A17-APEX2 overexpression in vivo. SLC6A17 was fused in-frame to three repeats of the HA tag, a V5 tag, and APEX2. (F) Representative EM image of synaptic vesicles (SVs) labeled by SLC6A17-APEX2 and darkness distribution of DAB-positive and DAB-negative SVs in sections of Slc6a17-APEX2 mouse brains. Red arrow pointing to APEX2 labeled SVs. White arrow pointing to unlabeled SVs.
Figure 4—source data 1. Original files of the full raw unedited blots for Figure 4C and D.
Figure 4—source data 2. Uncropped blots with the relevant bands labeled for Figure 4C and D.