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. 2023 Jul 19;14:1148684. doi: 10.3389/fimmu.2023.1148684

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Copyright © 2023 Li, Guo, Zhao, Kang, Lu, Long, Huang, Chen, Zhao, Wu, Yin and Li

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Leave-one-out sensitivity based on IVW model for Gut microbiota on Asthma and its phenotypes (A) Barnesiella; (B) CandidatusSoleaferrea; (C) RuminococcaceaeUCG014(Asthma); (D) Akkermansia; (E) Collinsella; (F) RuminococcaceaeUCG014(Childhood-onset asthma); (G) FamilyXIIIAD3011group; (H) Eisenbergiella; (I) Ruminiclostridium6. The IVW causal estimate and how the overall estimate (red horizontal line) was disproportionately driven, which is influenced by the removal of a single variant (black horizontal line), were visualized. There was no evidence of obvious heterogeneity, indicating that no specific SNP alone accounted for the association of gut microbiota on Periodontitis. The results suggested that there was no individual SNP with a strong influence on the overall effect. SNP, single-nucleotide polymorphism; IVW, Inverse variance weighted.