Table 5.

Histological characteristics of the 13 patients whose slides were reviewed for the purpose of this study and showed inflammation.

Demographics	Predominant injury pattern (acute/chronic)	Intraepithelial lymphocytes	LP inflammation (none/mild/moderate/severe)	Apoptosis (present/absent)	Lymphoid aggregates (absent/present)	Eosinophils
60/W/M	Chronic active gastritis with ulcer	Absent	Severe (plasma cell)	Absent	Absent	Present (rare)
74/W/F	Chronic gastritis with lymphocytic gastritis pattern	Present (in deeper glands and surface)	Mild (plasma cells)	Present	Absent	Present (rare)
74/B/M	Reactive gastropathy	Present (rare in deep glands)	Absent	Present	Absent	Absent
76/W/F	Reactive gastropathy	Absent	None	Absent	Absent	Absent
59/M/O	Focally enhanced gastritis	Absent	Moderate (plasma cells)	Present	Absent	Absent
75/W/M	Reactive gastropathy	Present (in deeper glands)	Mild (plasma cells)	Present	Absent	Absent
65/W/F	Lymphocytic Gastritis	Present	Moderate (plasma cell)	Present	Present	Present (rare)
50/W/M	Chronic inactive gastritis	Absent	Mild (plasma cells)	Absent	Absent	Absent
50/W/M	Chronic gastritis	Absent	Moderate (plasma cells)	Absent	Absent	Present
79/W/M	Chronic gastritis with focal IM	Absent	Moderate (plasma cell)	Absent	Present	Absent
51/W/M	Focally enhanced gastritis	Absent	Mild (plasma cells)	Absent	Absent	Present (rare)
43/W/F	Acute gastritis	Present	Moderate (neutrophilic and plasma cells)	Present	Absent	Present (rare)
69/W/M	Reactive gastropathy	Absent	Mild (plasma cells and eosinophils)	Present	Absent	Present