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. 2023 Aug 4;21:253. doi: 10.1186/s12951-023-02006-x

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 9 — Prediction of miRNA targets and gene silencing effect of miRNAs mediated apoptosis of tumor cells through the regulation of apoptotic c-MYC signaling pathway. a miRNA targets were predicted using miRNA target prediction tool based on sequence-based miRNA target prediction. b The binding site of ptc-miR396f in the mRNA of c-MYC. c Relative gene expression levels of c-MYC and BCL2 with miRNAs. There was a significant gene silencing effect compared to mock-transfected and control groups, which are normalized to non-targeting control (NTC) (*, P < 0.05; ***, P < 0.001)