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. 2022 Dec 18;10(3):358–378. doi: 10.1080/23328940.2022.2149024

Table 1.

Connecting personal heat exposure (PHE), which focuses on quantifying environmental heat load, with heat stress and strain to demonstrate the modeling goals within the current study. By leveraging the scope and definition of PHE to assess compensability under heat stress, we show how heightened PHE can lead to a rise in core temperature, dehydration, and cardiovascular strain over time.

PHE Definition “The realized contact between a person and an indoor or outdoor environment that poses a risk of increases in body core temperature, perceived discomfort, or both.” (Kuras et al.[2] p. 2) EnvironmentalheatloadPHEfTair,H,Ws,R,Pdt
*Note: Exposure itself implies intensity, duration, and frequency.
graphic file with name KTMP_A_2149024_ILG0001.jpg PHE is inherently linked to thermal stress and strain due to the coupling of exposure with duration and frequency to result in a meaningful exposure-response relationship.[9]
Heat Stress The net heat load on a person resulting from the combined thermal effects of the environment (air temperature, radiant temperature, humidity, and wind), metabolic heat production, and clothing.[68]
Heat Stress→f(Tair, H, Ws, R, P, M, Iclo)dt= PHE + M & Iclo
Heat Strain Effects on body physiology that occur as a consequence of heat stress[13]; can lead to a rise in body temperature due to body heat storage, dehydration from non-replenished sweat losses, and cardiovascular strain, such as heart rate increases to maintain blood pressure (Vanos et al[6]).
  • ‐ Incorporates physiologic factors to determine if heat stress is compensable or uncompensable over time


Heat Strain→(Tair, H, Ws, R, P, M, Iclo, Tsk, SR) = Heat stress + Tsk, , & SR

temperature (Tair), humidity (H), wind speed (Ws), radiation (R), atmospheric pressure (P), metabolic rate (M), clothing Insolation (Iclo), skin temperature Tsk, skin wettedness (ω), and sweat rate (SR).