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Effect of telomere dysfunction‐induced senescence on endothelium‐dependent and ‐independent vasodilation. (a) Dose–response curves to acetylcholine in mesenteric arteries in the absence and presence of the nitric oxide synthase inhibitor l‐NAME. (b) Dose–response curves to the exogenous nitric oxide donor sodium nitroprusside in mesenteric arteries. N = 13–17 per group. Data are mean ± SEM. *p < 0.05 versus EC‐TRF2f/f acetylcholine, # p < 0.05 versus acetylcholine without l‐NAME.
