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Tr-OxPLs induce more severe barrier disruption in lung ECs from old mice. ECs isolated from young and old mouse lungs were challenged with increasing concentrations (0–50 µg/mL) of indicated Tr-OxPLs, and TER was monitored for 25–30 h. Normalized resistance was plotted against time to determine endothelial permeability; n = 6. Comparative analysis of dose-dependent TER changes caused by each Tr-OxPL in lung ECs from young and old mice; n = 6.
