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Hypothetical illustration showing PAR2-independent and PAR2-dependent components of allergic lung disease in mice caused by co-exposure to MWCNTs and HDM allergens. MWCNTs enhance HDM allergen-induced eosinophilic inflammation through a STAT6-dependent mechanism that does not involve PAR2. However, PAR2 is postulated to promote airway fibrogenesis by increasing the expression of ARG-1
