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. 2022 Sep 22;39(4):1413–1431. doi: 10.1007/s10565-022-09767-5

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Treatment of CCL24 blocking antibody hindered angiotensin II–induced heart failure and cardiac hypertrophy. A M-mode echocardiography of left ventricular chamber and the measurement of ejection fraction (EF %) and fractional shortening (FS %). The data are represented as the means ± SEM (n = 16; one-way ANOVA; P < 0.05). B Hematoxylin and eosin staining of heart section. Scale bar in panel A is 1000 μm. C Representative heart size, and heart weight to body weight (HW/BW) ratio. The data are represented as the means ± SEM (n = 6; one-way ANOVA; P < 0.05). D TRITC-labelled wheat germ agglutinin staining of heart tissue sections, and quantification of cardiac-myocyte cross-sectional area (50 cells counted per heart). Scale bar in panel C is 50 μm. The data are represented as the means ± SEM (n = 6; one-way ANOVA; P < 0.0001)