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. Author manuscript; available in PMC: 2024 Aug 1.
Published in final edited form as: Alzheimers Dement. 2023 Feb 15;19(8):3339–3349. doi: 10.1002/alz.12956

Figure 3: Correlation between Genetic Status and CSF Cell Counts.

Figure 3:

Correlation between ApoE4 genetic status and CSF cell populations (A-D). No significant correlation was observed between the ApoE4 genetic status and (A) CSF monocyte population (n=131), (B) CSF lymphocyte population (n=131), or (C) CSF neutrophil population (n=62). There was not a significant correlation between the ApoE4 genetic status and the (D) CSF monocyte-to-lymphocyte ratio (n=131).

Correlation between genetic status for monogenic disease genes (carrier vs non-carrier) and CSF cell populations (E-H). No significant correlation was observed between the presence of monogenic disease mutations and (E) CSF monocyte population (n=132), (F) CSF lymphocyte population (n=132), or (G) CSF neutrophil population (n=63). There was not a significant correlation between the presence of monogenic disease mutations and the (H) CSF monocyte-to-lymphocyte ratio (n=132).