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. 2023 Jan 3;11(1):449–463. doi: 10.1016/j.gendis.2022.12.003

Figure 6.

Fig. 6

CD36 regulates ferroptosis via FSP1 in AKI. (A) After CD36 overexpression or iFSP1 treatment, western blotting was used to detect the expression of CD36, FSP1, and ACSL4 in TCMK-1 cells, and quantitative statistical analysis for each protein (n = 3) was also performed. Data are expressed as means ± standard deviation. ∗P < 0.05 and ∗∗P < 0.01 vs. control group. (B) Mechanism diagram. Ferroptosis was inactivated in cells after AKI, resulting in CD36 overexpression, which promoted AKI after the ubiquitination of FSP1.