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. 2023 Aug 14;21:203. doi: 10.1186/s12964-023-01193-2

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Diabetes mellitus promotes the progression and TME remodeling in CC. a Clinical characteristic were analyzed between CC patients with DM and without DM and genes on TME biological diversity were detected by human mRNA-microarray, respectively. b, c Vesicle-related genes were determined to link to EMT and/or EndMT through Pearson correlation analysis and GENEMANNIA database. d Volumes of xenograft were assessed in mice with and without DM. CC: colon cancer, DM: diabetes mellitus, MOS: months. Data are analyzed using unpaired student’s t-test and χ² test. *p < 0.05