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. 2023 Jul 26;620(7974):607–614. doi: 10.1038/s41586-023-06333-9

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Extended Data Fig. 3 — a, Representative haematoxylin and eosin staining of normal lobules, benign breast lesions (BBL), and cancer lesions (top row), and the corresponding images at a high power magnification (bottom row) (14 out of 337 lesions). b, Representative LCM images before and after the dissection (one out of 194 lesions). c, Representative sequence of lesions originating from the same clones (four out of seven clones). Founder driver alterations in each clone are shown in black. Scale bar = 150 μm.