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. 2023 Jun 6;11(3):125–134. doi: 10.1055/s-0043-57266

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The Author(s). This is an open access article published by Thieme under the terms of the Creative Commons Attribution-NonDerivative-NonCommercial License, permitting copying and reproduction so long as the original work is given appropriate credit. Contents may not be used for commercial purposes, or adapted, remixed, transformed or built upon. ( https://creativecommons.org/licenses/by-nc-nd/4.0/ )

This is an open-access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivatives License, which permits unrestricted reproduction and distribution, for non-commercial purposes only; and use and reproduction, but not distribution, of adapted material for non-commercial purposes only, provided the original work is properly cited.

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Fig. 1 — Venn diagram of pathways and genes associated with ascending thoracic aortic aneurysm (ATAA) and with abdominal aortic aneurysm (AAA). Note: Common genes are displayed in central overlap zone. RAAS: renin–angiotensin–aldosterone system. ‘‘*’' denotes the association of a gene with a known genetic syndrome (see Table 2 ). ‘‘Sporadic’' denotes the association of a gene, or group of genes, with sporadic ATAA. Unless otherwise stated, all ATAA genes are associated with hereditary ATAA. Genes with an uncertain AAA association: Genes for which more studies (e.g., genome-wide association studies and meta-analyses) need to be undertaken to establish a strong level of significance to sporadic AAA.