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. 2023 Aug 24;14(8):554. doi: 10.1038/s41419-023-06086-3

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — A Cross-sectional histological images showing changes in retinal morphology in DBA/2J-Atg4b^ko mice, including increased thickness of the INL and OS, and disorganization of the OS and IS (higher magnification shown in SM, Fig. 2). Images are representative of at least 6 animals per group. OS outer segment, IS inner segment, ONL outer nuclear layer, OPL outer plexiform layer, INL inner nuclear layer, IPL inner plexiform layers, GCL ganglion cell layer. B Ultrastructural images of the GCL showing the higher RGC density (asterisk, representative) and the absence of apoptotic and necrotic bodies (arrows) with deletion of Atg4b.