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. 2023 Aug 21;24(16):13017. doi: 10.3390/ijms241613017

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© 2023 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Therapeutic mechanism of miR-25-5p, miR-214-3p, miR-142-5p, miR-340-5p, miR-26a-5p, and miR-490-3p. Bax: BCL2-associated X; bcl-2: B-cell lymphoma-2; CTGF: connective tissue growth factor; CTSB: cathepsin B; GSH: glutathione; IL-6: interleukin-6; IRAK1: interleukin 1 receptor-associated kinase 1; MAPK: mitogen-activated protein kinase; MDA: malondialdehyde; miR: microRNA; MyD88: myeloid differentiation factor 88; NF-κB: nuclear factor kappa-light-chain-enhancer of activated B cells; NLRP3: NOD-like receptor thermal-protein-domain-associated protein 3; ROS: reactive oxygen species; SOD: superoxide dismutase; TNF-α: tumor necrosis factor; TRAF6: TNF receptor-associated factor 6; TXNIP: thioredoxin-interacting protein.