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Emma Lewis et al. (5) demonstrate the ability of pregnancy to induce a hypofunctional state in fetus-specific CD8+ T cells. IFNγ, interferon-g; IL7R, interleukin-7 receptor, TNFα, tumor necrosis factor-α; Tox, PD-1, Lag3, CD38, co-inhibitory markers associated with T cell dysfunction and exhaustion; Satb1, CCR7, IL7-R, markers associated with typical effector T cell response
