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. Author manuscript; available in PMC: 2023 Aug 31.
Published in final edited form as: Pflugers Arch. 2021 Feb 26;473(3):351–362. doi: 10.1007/s00424-021-02538-x

Fig. 3.

Fig. 3

Changed abundance of CSQ proteins in ischemic failing human hearts. a Immunoblotting images of β-actin, calsequestrin (CSQ), and GAPDH in right atria of donors with of ischemic heart failure (Isch-HF) compared to that of aged-matched controls (CTL); b immunoblotting images of β-actin, CSQ, and GAPDH in the left ventricle of donors with ischemic heart failure (Isch-HF) compared to that of aged-matched controls (CTL); c–e summarized data of β-actin (c), CSQ (d), and GAPDH (e) protein expression in right atria (left panel) and left ventricle (right panel) of human organ donors with ischemic heart failure (Isch-HF) compared to that of aged-matched non-failing controls (CTL). Data are represented as mean ± SEM. *p < 0.05; **p < 0.01. Isch-HF RA, n = 6; non-HF CTL RA, n = 7; Isch-HF LV, n = 7; Non-failing CTL LV, n = 5