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. 2023 Aug 30;14:231. doi: 10.1186/s13287-023-03455-9

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — IL-1β in the serum of ACLF patients can increase the level of IL-8 in hUC-MSCs. A Effects of TNF -α, IL-1β, LPS and IFN-γ on the expression of IL-8 in hUC-MSCs. B The expression levels of IL-1β and TNF-α in the serum of different groups were detected by ELISA. C After IL-1β stimulation of hUC-MSCs for 24 h, changes in the pathways related to IL-8 expression in hUC-MSCs were detected by Q-PCR. D The effect of IL-1β on the NF-κB signaling pathway and IL-8 expression in hUC-MSCs was detected by Western blotting. Full-length blots are presented in supplementary figure s2. *P < 0.05; **P < 0.01; ***P < 0.001 (DPI:300)